Intended for healthcare professionals

Rapid response to:

News Extra [these Stories Appear Only On The Web]

Pathologist in Sally Clark murder case is charged with withholding vital evidence

BMJ 2005; 330 doi: https://doi.org/10.1136/bmj.330.7485.216-b (Published 27 January 2005) Cite this as: BMJ 2005;330:216

Rapid Response:

Re: Re: Re: A Fatal Misdiagnosis

Dr Innis writes in his rapid response to “Pathologist in Sally Clark
murder case is charged with withholding vital evidence” [Dr Williams
withheld the finding of a ubiquitous bacterium Staphylococcus aureus in
many sites of the body of one of the babies, including the CSF] that “I
for one, like Dr Williams would not have reported such a finding as it is
clearly a postmortem artifact.” Quite understandably this met with a
strong response from Mr Lockyer, Sally Clark’s father, since she was freed
on appeal based on this finding.

Dr Innis continued that “The GMC, by using it to prosecute Dr
Williams, is avoiding the real issue which is the role of vaccines in the
death of the child.”

So, what’s the truth? Was it vaccination or was it the bacterial
infection? The former was the underlying cause, the latter was the final
and visible pathway to death.

It is a well documented fact in orthodox medical literature that
vaccines suppress the immune system and make their recipients susceptible
to infections with a host of bacteria (including Staphylococcus aureus)
and viruses. This has been especially well researched in the case of DPT
and Hib vaccines both of which were given to both of Sally Clark’s babies.
Craighead (1975, J infect Diseases; 131(6): 749-754) wrote, “In 1960 Cox
reported that animals immunized with several low-potency, inactivated
viral and rickettsial vaccines responded to challenge with an infection of
increased severity… Subsequently, several groups of investigators
documented the sporadic occurrence of an atypical pattern of naturally
acquired measles several years after the administration of an inactivated
virus vaccine to children [3-5]. In 1967 Smith et al. described a febrile
illness accompanied by pneumonia in experimentally infected recipients of
killed Mycoplasma pneumoniae vaccine who failed to produce detectable
antibody [6]… Kim et al. [8], Fulginiti et al. [9] and Chin et al. [10]
simultaneously reported an unusually severe respiratory disease in
children developing natural infections with respiratory syncytial virus
after immunization with formaldehyde-treated vaccine. These observations,
although limited in scope, suggested that immunization with inactivated
vaccines could “sensitise” the recipient and result in an accentuated
pattern of disease upon natural or experimental exposure.”

Daum et al. 1989, J Pediatrics; 114: 742-747 described decline in
serum antibody to the capsule of Haemophilus influenzae type b in the
immediate “post immunization” period.

Researchers from University of Minnesota (Minneapolis, USA)
researched antigen-induced transient hypersusceptibility as a cause of
sporadic and fulminant (meaning rapid, sudden, severe) infection in normal
infants. They established clustering along certain days “post
immunization”. They also stated “Others have documented profound
alterations in T-cell ratios and decreased responsiveness in 2nd week post
immunisation, coinciding with period of hypersusceptibility noted here in
humans.” Jefferys (Lancet 2001;357:1451) described more than one process
by which vaccination is likely to derange the T-cell homeostasis, which
would result in immunosuppression (and autoimmunity). There does not have
to be any signs of local inflammation.

There is no doubt about the effect of hypersusceptibility in the
minds of those who have published their research on the deleterious
effects of vaccines on the immune system, which in some babies leads to
fulminant infection being the final pathway to death. Both Clark baby boys
died after vaccination (one a few hours, the other about 3 weeks, which
are the critical times after vaccination, with increased numbers of
deaths), but the final direct cause of their death was fulminant
infection.

There is no need for any conflict.

Competing interests:
None declared

Competing interests: No competing interests

17 February 2005
Dr Viera Scheibner
Principle Research Scientist (Retired)
Blackheath, NSW 2785 Australia