What's new in the other general journals
BMJ 2005; 331 doi: https://doi.org/10.1136/bmj.331.7507.13 (Published 30 June 2005) Cite this as: BMJ 2005;331:13All rapid responses
Rapid responses are electronic comments to the editor. They enable our users to debate issues raised in articles published on bmj.com. A rapid response is first posted online. If you need the URL (web address) of an individual response, simply click on the response headline and copy the URL from the browser window. A proportion of responses will, after editing, be published online and in the print journal as letters, which are indexed in PubMed. Rapid responses are not indexed in PubMed and they are not journal articles. The BMJ reserves the right to remove responses which are being wilfully misrepresented as published articles or when it is brought to our attention that a response spreads misinformation.
From March 2022, the word limit for rapid responses will be 600 words not including references and author details. We will no longer post responses that exceed this limit.
The word limit for letters selected from posted responses remains 300 words.
Could Dr Flachsbart elaborate on his comments concerning a possible infectious aetiology of
diabetes?
This would be of special interest to me as I have harboured a
suspicion along those lines (with a {perhaps} unrelated disease) for a few
years.
Competing interests:
None declared
Competing interests: No competing interests
Dear Sir,
the monoclonal antibodies lowered the inflammatory response to a
hidden antigen.
In consequence the infection became observable:
Sore throat and cervical lymphadenopathy.
Prof. Veil, Jena, prophecied this 1939:
Streptococcal sore throat is a cause of diabetes.
A study with monoclonal antibodies and penicillin versus penicillin
should test therapy versus placebo.
Sincerely Yours
Friedrich Flachsbart
Competing interests:
None declared
Competing interests: No competing interests
Re: Re: Sore throat: The hidden cause of diabetes unveiled.
Dear Sir,
Dr. Nehrlich asked for elaboration on a possible infectious aetiology
of diabetes.
Obesity and diabetes are like gout associated with a state of chronic
low-level inflammation.
The inflammation is survival-benefit against infection, especially
against rheumatic fever, rheumatic heart disease and post-streptococcal-
reactive-glomerulonephritis.
In my praxis common cold and streptococci are the pathogen triggering
the inflammation of these metabolic diseases.
There is no animal model like the human suffering from disease.
Sincerely Yours
Friedrich Flachsbart
KE Wellen, GS Hotamisligil:
Inflammation, stress, and diabetes.
J Clin Invest. 2005;115:1111-9
BO Roep, M Atkinson, M. von Herrath:
Satisfaction (not) guaranteed: Reevaluating the use of animal models of
Type 1 Diabetes.
Nature Reviews Immunology 2004;4:989-997
Competing interests:
None declared
Competing interests: No competing interests