BMJ 2008; 337 doi: https://doi.org/10.1136/bmj.a1115 (Published 05 August 2008) Cite this as: BMJ 2008;337:a1115

Minerva hangs her head in shame after casting undeserved aspersions on neurologists a few weeks ago (doi 10.1136/bmj.39597.485556.47). Misquoting a study in Neurology, she managed to identify neurologists as the most disruptive group of non-surgical specialists. It was actually cardiologists who were deemed to have the most disruptive behaviour, with gastroenterologists and neurologists coming a close joint second. Minerva wishes to apologise to non-disruptive neurologists everywhere.

Animal experiments indicate that refined carbohydrates contribute to the development of non-alcoholic fatty liver disease. Carbohydrates such as fructose can cause intestinal bacterial overgrowth and increased intestinal permeability, which ends up creating a fatty liver. Plasminogen activator inhibitor (PAI)-1 has also been linked to liver damage. Now a pilot study in the Journal of Nutrition (2008;138:1452-5) has compared dietary factors, endotoxin, and PAI-1 concentrations in patients with non-alcoholic fatty liver and controls. It found that dietary fructose, increased intestinal translocation of bacterial endotoxin, and PAI-1 may all contribute to formation of fatty liver in humans.

A review in Clinical Diabetes (2008;26:115-20) offers tips to help patients who are trying to lose …

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