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Published 5 August 2009, doi:10.1136/bmj.b2920
Cite this as: BMJ 2009;339:b2920
Luis Corral-Gudino, internal medicine consultant1, Ramón J Jorge-Sánchez, internal medicine consultant1, María Borao-Cengotita-Bengoa, internal medicine trainee1, Judit García-Aparicio, internal medicine consultant 1, Manuel Cascón-Bueno, cardiology consultant2
1 Department of Internal Medicine, Hospital los Montalvos, Hospital Universitario de Salamanca, 37192, Salamanca, Spain , 2 Department of Cardiology, Hospital Universitario de Salamanca, 37007, Salamanca, Spain
Correspondence to: L Corral-Gudino lcorral@husa.sacyl.es
| The first 150 words of the full text of this article appear below. |
A 75 year old woman was admitted to hospital because of dyspnoea. She had been well until nine months previously, when she started to have gradually worsening exertional dyspnoea. On admission, her physical activity was limited. She had no discomfort at rest, but even a very low level of activity caused dyspnoea. She reported no chest pain, orthopnoea, or paroxysmal nocturnal dyspnoea. She had a history of systemic hypertension. She denied smoking. Physical examination showed that she had tachypnoea and a tricuspid systolic murmur. Lung sounds were normal. Electrocardiography showed right axis deviation, right ventricular hypertrophy, and atrial fibrillation. Laboratory evaluation showed polycythaemia. Examination of her chest radiograph at admission provided clues to the diagnosis (fig 1)
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