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Published 6 May 2009, doi:10.1136/bmj.b1809
Cite this as: BMJ 2009;338:b1809
| The first 150 words of the full text of this article appear below. |
Monocular transient visual loss due to emboli or ocular hypoperfusion usually occurs in isolation rather than accompanying a transient neurological deficit.1 Adhiyaman and Adhiyaman did not mention that giant cell arteritis is an important cause of transient visual loss in their 10 minute consultation on transient ischaemic attack.2 Transient visual loss occurred in over 30% of patients with giant cell arteritis and ophthalmic involvement in one series and is a strong predictor of permanent visual loss.3 4 Delayed diagnosis and treatment of giant cell arteritis is associated with bilateral blindness in patients with visual symptoms.4
The authors also do not mention measuring inflammatory markers in the recommended investigation of transient ischaemic attack,2 but we would advise checking erythrocyte sedimentation rate, C reactive protein, and platelet count in older patients with transient monocular visual loss. High dose glucocorticoid treatment (prednisolone 1 mg/kg, maximum 60 mg, or pulsed methylprednisolone) should be started before
Catherine M Guly, specialty registrar in medical ophthalmology1, John A Olson, consultant ophthalmic physician1
1 Aberdeen Royal Infirmary, Aberdeen AB25 2ZN
catherine.guly@doctors.org.uk